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Legionella effectors, including SidF, inhibit host apoptosis by antagonizing pro-apoptotic Bcl-2 family proteins, allowing prolonged intracellular survival ( 3.4 Mitochondrial ROS and metabolic reprogramming in bacterial infection Bacterial infections frequently induce mtROS as a byproduct of disrupted ETC function or calcium overload

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Chronic treatment with fluoxetine decreases cerebral metabolic responses to the 5-HT1A agonist 8-hydroxy-2(di-N-propylamino)tetralin and increases those to the 5-HT2A/2C agonist 1-(2,5-dimethoxy-4-iodophenyl)-2-aminopropane and to the dopaminergic agonist apomorphine

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