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This review integrates mechanistic insights with translational advances, outlining how dysregulation of chaperones, autophagy-mitophagy, the ubiquitinproteasome system, and ER stress pathways drive human diseases, while highlighting emerging therapeutic platforms, from pharmacological chaperones and autophagy modulators to targeted protein degradation technologies, CRISPR screens, spatial biology, and AI-guided drug discovery

These changes may occur in months rather than years, creating the impression of sudden ageing

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The emergence of single-cell and single-nucleus transcriptomics revealed previously hidden heterogeneity and enabled trajectory-level analysis of MuSC fate dynamics