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Therefore, our observations of both the reduced mobility of G3BP in cells expressing the VCP bindingdefective mutant and the ASPL-mediated stabilization of the G3BP-VCP interaction in cells subjected to either SA or HS suggest a role for the ASPL-VCP interaction in the VCP-mediated extraction of G3BP independent of G3BPs ubiquitination status

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5F, protein banding patterns of MCMs and SP@M were nearly identical according to the Coomassie blue staining assay, demonstrating the effective translocation of MCMs proteins onto the SP substrates

One meta-analysis reported a statistically significant increase in risk, whereas the remaining seven showed no significant associations

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