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Investigations into the mechanisms involved in OTA-induced toxicities have included studies on its accumulation in target organs, biotransformation in several species, and its potential role in oxidative stress (Kszegi and Por 2016

123 Besides, oxidative stress conditions also increase the innately high IDH1 expression, and IDH1 silencing significantly enhances cell sensitivity to cancer chemotherapy, radiotherapy, and photodynamic therapy by reducing NADPH

An aminoisobutyric acid (Aib) substitution at position 8 protects against degradation by dipeptidyl peptidase-4 (DPP-4), while attachment of a C18 fatty diacid chain via a -glutamic acid spacer promotes strong binding to serum albumin

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