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Opportunities and challenges in the discovery of allosteric modulators of GPCRs for treating CNS disorders

sCT alone and combined with liraglutide treatment increases the number of cFos- activated neurons in the DVC of adult rats In an attempt to identify nuclei within the brain that may offer a complementary set of neural circuits that mediate, at least in part, the combined anorectic effects of amylin and GLP-1 pharmacotherapy, we focused our attention on those nuclei in the DVC given the wealth of literature showing the contribution of this structure to the intake suppressive effects of either amylin- or GLP-1-based monotherapy 6,7,8,10

Inactive (depolarized) mitochondria decrease MMP

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