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doi: 10.1111/mcn.12522 27

The elevated calcium levels also impair mitochondrial function, triggering the release of proapoptotic factors, including cytochrome c, and activating caspase-dependent apoptotic pathways ( tThe excitotoxicity induced by METH also triggers neuroinflammation and amplifies its neurotoxic effects

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Loureirin C inhibits ferroptosis after cerebral ischemia reperfusion through regulation of the Nrf2 pathway in mice