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The impaired interaction decelerated the oxidative half-reaction and decreased the steady-state concentration of available glutathionylated enzyme for the rate-limiting turnover of GSH

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Last updated: March 22, 2026 Introduction to Inflammatory Bowel Disease The etiology of Inflammatory bowel disease (ulcerative colitis and Crohns disease) is complex, with genetic predisposition, an altered microbiome, environmental factors and a weakened epithelial (gut) barrier function triggering a chronic immune response in the mucosal layer

HG-induced iron overload generates hydroxyl radicals, causing mitochondrial membrane potential (MMP) depolarization, mtDNA damage, and dysfunction (139142)