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Interestingly, it was previously proposed that the ability of KEAP1 to sense H 2 O 2 depends on the formation of a disulfide bond between Cys 226 and 613, and this sensing mechanism differs from that used by electrophilic NRF2 inducers, suggesting the plausibility of cysteinylation of these two residues in cells [25]

DATS reduced the incidence of poorly differentiated prostate tumors and limited the number of metastatic lesions in the lungs of mice genetically modified to develop prostate adenocarcinomas (71)

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Mitochondrial cascade hypothesis Mitochondrial dysfunction and the accumulation of reactive oxygen species lead to neuronal damage and ultimately contribute to the pathogenesis of neurodegenerative diseases, particularly AD