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Fasting is required for CR-induced improvements in tau hyperphosphorylation and neuroinflammation, but not for CR-induced improvements in A plaque deposition To investigate the role of fasting in the ability of CR to attenuate AD pathology, we evaluated several pathological hallmarks of AD, including phosphorylation of tau, amyloid beta (A) plaque deposition, and gliosis

These diseases may affect more people in todays aging society

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Wedelolactone from Eclipta alba inhibits lipopolysaccharide-enhanced cell proliferation of human renal mesangial cells via NF-B signaling pathway