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Similarly, patients with conflicting data on the key criteria of fatigue, unrefreshing sleep, and postexertional malaise, who could not be dependably identified from documentation thereafter, were excluded (13, 17, and 55 patients, respectively)

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These limitations have driven decades of research into smaller molecules that can activate TrkB signaling with better pharmacokinetic properties

Mechanistically, CPT1A restrains ubiquitination and degradation of c-Myc, while c-Myc transcriptionally activates CPT1A expression