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For instance, studies combining metagenomics, metatranscriptomics, and metabolomics in prodromal and established PD have identified early functional alterations in microbial activity, including changes in metabolites such as isovalerate and valerate, and shifts in pathways related to glutamate metabolism, chemotaxis, and flagellar assembly [19]

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Abnormal expression of these two families is primarily a result of gene dysregulation and translocation from organelles, which result in tumorigenesis mainly through two ways, the regulation of downstream molecular targets and the unsteady state of zinc homeostasis

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