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Reduced prefrontal synaptic connectivity and disturbed oscillatory population dynamics in the CNTNAP2 model of autism

5-Amino-1MQ and MOTS-c both reduced fat mass in rodent studies through NAD+-linked and AMPK-linked mechanisms [2][5]

Mutations in the KCNJ11 gene are associated with MODY13, transient neonatal diabetes mellitus type 3 (TNDM3), permanent neonatal diabetes mellitus (PNDM), familial persistent hyperinsulinemic hypoglycemia of infancy (PHHI), and also contributes to an autosomal dominant form of type 2 diabetes

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VEGF itself is a potent inducer of eNOS expression, and NO in turn stabilizes VEGF mRNA against degradation a positive feedback loop that amplifies angiogenic signaling