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Wherein, the phosphorylation of the C-terminus by ERK1/2 recruits -arrestins to sterically hinder interactions with GPCR kinases, initiating endocytosis likely via the assembly of clathrin-coated pits [24, 33]

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This dual stimulation results in a more robust and sustained release of growth hormone, mimicking the natural, youthful rhythm of secretionsomething no single peptide can achieve alone

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