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Inactivation of S1PR2, for example, a sphingosine receptor which also binds to Nogo-A preventing axonal growth, resulted in increased remyelination in both lysolecithin-induced demyelination and EAE mice (59), further exemplifying the potential role of S1P and its analogues
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Ijima, S
SSG of mitofusin 2 (MFN2) disrupts mitochondriaendoplasmic reticulum contact sites, compromising organelle communication and promoting activation of the RIPK1-RIPK3-MLKL axis in neurotoxicity models 82
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