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One possible explanation is that low serum vitamin B12 levels would trap folate as 5-methyltetrahydrofolate, prevent the generation of methionine from homocysteine, and therefore reduce protein synthesis and lean tissue deposition

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BPC-157 mainly targets gut and connective-tissue repair through NO and VEGF signaling, while TB-500 (a Thymosin Beta-4 analog) drives repair through actin and new blood vessel growth

Parasites can also be a trigger for MCAS given that mast cells are one of our protective mechanisms for parasites

*Correspondence: Boutaina Daher [email protected] Pouyssgur [email protected] This article was submitted to Cancer Metabolism, a section of the journal Frontiers in Oncology Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers