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The results showed that allicin significantly reduced the expression of cleaved caspase-3 in mice with cerebral infarction, but no significant effect was observed in the sham operation group

Epigenetic profiling further demonstrated an age-associated accumulation of repressive H3K27me3 marks and the formation of bivalent chromatin domains, which limit the expression of regenerative genes and contribute to MuSC dysfunction

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This makes it challenging to separate drug effects from underlying disease risk in research studies

Increasing evidence implicates insulin resistance (IR) as a key pathophysiological factor linking metabolic and psychiatric illness